Putting rats on total parenteral nutrition (e.g., intravenous feeding) causes the monooxygenase receptor for vitalethine to plummet to only a fourth or fifth of its normal level. Supplementing with an “alcoholic extract of rat chow (food)” completely reversed this effect. When prepared similarly, “rat chow tincture” analyzed at LANL, showed the majority of the material extracted was the same size as N, N-dimethylglycine, a component (with gluconic acid) of pangamic acid (vitamin B15). Thus, toxic metal poisonings may be offset by dimehtylglycine that increases the monooxygenase receptor for vitalethine, with an associated stabilizing effect of the enzyme upon vitalethine and vice versa. Pangamic acid is the natural, “raw” form of dimethylglycine.